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Weck, Jennifer

Formal Title:

Scientific Program Specialist


Jennifer Weck is a Scientific Program Specialist with the Division of Epidemiology, Statistics, and Prevention Research at NICHD. In this capacity, she provides expertise regarding laboratory techniques and interpretation of results, chairs the Biospecimen Repository Access and Datasharing (BRADS) Committee, and manages NICHD’s Biorepository.





6710B ROCKLEDGE DRIVE Room 3237C, MSC 7004
Bethesda ,MD 20817

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 Dr. Weck received a B.S. from Duke University and her Ph.D. from the Department of Molecular Physiology and Biological Physics from the University of Virginia where she worked with Margaret Shupnik studying transcriptional regulation of pituitary genes involved in female reproduction.   She completed a postdoctoral fellowship at Northwestern University under the guidance of Kelly Mayo and was a Research Assistant Professor at the Northwestern Feinberg School of Medicine as well as at Washington State University prior to coming to the NIH. 


Publications (PubMed):

Switching of NR5A proteins associated with the inhibin alpha-subunit gene promoter after activation of the gene in granulosa cells.
Role of the phosphatidylinositol-3-kinase and extracellular regulated kinase pathways in the induction of hypoxia-inducible factor (HIF)-1 activity and the HIF-1 target vascular endothelial growth factor in ovarian granulosa cells in response to follicle-stimulating hormone.
Follicle-stimulating hormone/cAMP regulation of aromatase gene expression requires beta-catenin.
Sequence-specific deoxyribonucleic acid (DNA) recognition by steroidogenic factor 1: a helix at the carboxy terminus of the DNA binding domain is necessary for complex stability.
Membrane-Type 6 Matrix Metalloproteinase Regulates the Activation-Induced Downmodulation of CD16 in Human Primary NK Cells.
Lhcgr Expression Granulosa Cells: Roles for PKA-Phosphorylated ß-catenin, TCF3, and FOXO1.
Toso, a functional IgM receptor, is regulated by IL-2 in T and NK cells.
Complex regulation of human NKG2D-DAP10 cell surface expression: opposing roles of the ¿c cytokines and TGF-ß1.
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